This page describes pharmacological agents that may have legal restrictions, side effects, and drug interactions in your jurisdiction. Information is for educational research only — consult a clinician before considering any compound.

Browse

Phenylpiracetam

Well Researched

Russian-developed phenyl-substituted piracetam analog created in 1983 for Soviet cosmonauts — the only racetam on the WADA prohibited list…

Aliases (15)
Phenotropil · Phenotropil® · Phenotropyl · Carphedon · Carfedon · Fonturacetam · Fonturacetamum · Nanotropil Novo · Nanotropil Novo® · Aktitropil · Aktitropil® · Entrop · 4-Phenylpiracetam · (RS)-2-(2-oxo-4-phenylpyrrolidin-1-yl)acetamide · PHENYLPIRACETAM
TYPICAL DOSE
100 mg
BID
ROUTE
Oral (tablet)
Oral
CYCLE
PRN only
As prescribed
STORAGE
Room temp; original container
Room temp

Overview

What is Phenylpiracetam?

Phenylpiracetam (Phenotropil, Carphedon) is a Russian-developed pyrrolidinone nootropic, a phenylated derivative of piracetam with markedly enhanced potency, lipophilicity, and stimulant-like cognitive effects. It is registered in Russia for cognitive disorders and is banned by WADA.

Key Benefits

Reported to enhance focus, motivation, physical performance, cold tolerance, and learning, with a stimulant-like subjective profile but without classic catecholamine release. Tolerance develops rapidly to subjective effects, limiting daily use.

Mechanism of Action

Modulates AMPA glutamate receptors, NMDA receptors, and nicotinic acetylcholine receptors. Increases dopamine, norepinephrine, and serotonin levels and enhances dopamine D1/D2 receptor density. Inhibits dopamine reuptake to a mild degree.

Pharmacokinetics

·
PeakHalf-life
Approximate curve — visual aid only, not data-precise PK
Brand options8 known
PhenotropilPhenotropylCarphedonCarfedonFonturacetamFonturacetamumNanotropil NovoAktitropil

StatusUnscheduled in US (legal gray-market import for personal use). Rx-only in Russia (registered as Phenotropil 2003; off-market 2017-2018; reissued as Nanotropil Novo® and Aktitropil® by Pharmstandard ~2020+). WADA-banned in-competition since **1998** (S6 stimulant) — this is the date in WADA prohibited-list documentation; some sources misstate as 2002, which is when high-profile athlete cases began under the new code.

Peptide Interactions

alpha-gpc
Synergistic

/ citicoline (the user's V stack includes 500 mg citicoline daily): Mandatory pairing to prevent racetam headache and amplify cognitive effect. Pre-dose by 3…

l-tyrosine
Synergistic

Theoretical synergy via increased DA substrate when reuptake is blocked. Anecdotal reports mixed but mostly positive. Try 500-1000 mg with phenylpiracetam dose.

pramiracetam
Synergistic

Different mechanism (HACU enhancement vs. DAT inhibition), no cross-tolerance reported. Can be used on alternate days — pramiracetam for sustained daily cogn…

sulbutiamine
Synergistic

Similar PRN profile, complementary mechanism (thiamine derivative, mild DA via different pathway). Can stack on the same PRN day for combined drive + cogniti…

modafinil
Synergistic

Different DAT-binding kinetics; functionally synergistic for wakefulness + drive, but don't stack same-day on a routine basis — phenylpiracetam should be a *…

L-theanine
Synergistic

(the user's V stack has 200 mg): Smoothing partner — softens any mild irritability/agitation, no efficacy compromise.

Magnesium
Synergistic

(the user's V stack): Neutral-positive; helps sleep onset on phenylpiracetam days if dosed PM.

Creatine
Synergistic

(users in this archetype often already take): Synergistic for the physical-performance arm; both improve high-output cognitive + physical work.

Beta-alanine
Synergistic

(the user's V stack): Synergistic for endurance / training-day applications.

Other DAT inhibitors same-day
Avoid

(modafinil daily-stack, amphetamine, methylphenidate, bromantane): additive sympathetic + DA tone, hypertension/anxiety/sleep risk. Pick one per day.

High-dose caffeine
Avoid

(>200 mg): additive stimulation, jitter risk. Low caffeine (50-100 mg) is fine.

Phenibut
Avoid

not a pharmacological interaction concern, but the GABA-B agonism + stim lift produces a "rollercoaster" subjective profile most users dislike. Skip.

Quality Indicators

Pharmacy-dispensed, intact packaging

Prescription tablets in original sealed packaging from a licensed pharmacy.

!

Generic vs branded

Generics are usually fine but bioavailability can vary slightly; track if you switch.

Unbranded blister or counterfeit risk

Counterfeit pharmaceuticals are a known issue; verify pharmacy and lot if buying internationally.

What to Expect

  • Day 1
    PK-driven acute peak per administration. Verify dose tolerated.
  • Week 1
    Steady-state reached for most daily-dosed pharma.
  • Week 2-4
    Therapeutic effect established; titration window if needed.
  • Long-term
    Periodic monitoring per drug class (labs, BP, ECG as applicable).

Side Effects & Safety

  • Common (>10% users):
    • Insomnia / sleep onset delay if dosed past mid-afternoon — most common cause of bad outcomes; resolves with AM-only dosing.
    • Mild headache in choline-undosed users — fully preventable with co-dosed alpha-GPC or CDP-choline.
    • Mild irritability / agitation — typically dose-dependent, more common at 200+ mg, often resolves with food + L-theanine co-dose.
    • Tolerance buildup — universal at >5 consecutive days of daily use, partial within 1-2 weeks. Functionally a side effect because it forces cycling.
  • Less common (1-10%):
    • Mild appetite suppression (DAT inhibitor profile, similar to modafinil but milder)
    • Mild HR / BP elevation (modest sympathetic activation, smaller magnitude than modafinil)
    • Mild GI discomfort if dosed on empty stomach in sensitive users
    • Dry mouth
    • Mild flushing / warmth (transient, especially first doses)
    • Psychomotor agitation / "wired" feeling at higher doses (>200 mg)
  • Rare-serious (<1% but worth knowing):
    • Hypertension at supratherapeutic doses — documented in Russian clinical literature at >400 mg/day. Not a concern at 100-200 mg.
    • No documented cases of dependence, addiction, or withdrawal in the human literature. Tolerance is functional, not addiction-pattern.
    • Theoretical seizure threshold concerns — like piracetam, phenylpiracetam may lower seizure threshold in susceptible individuals. Not contraindicated in healthy users; caution if any seizure history.
    • No documented hepatotoxicity (consistent with non-hepatic metabolism).
    • No documented cardiac arrhythmia signal at therapeutic doses.
  • Specific watch periods:
    • Days 1-7 of any new pulse: monitor for excessive HR/BP if normally stim-sensitive; monitor sleep quality (afternoon dosing kills sleep).
    • Beyond 14 days continuous: tolerance is the main effect, not a safety signal — but you'll be wasting product. Cycle off.
    • Pediatric / under-18: Russian pediatric use exists but Western consensus is to avoid in developing brains. a 20-year-old in this archetype is past the highest-risk window but still in the "favor lowest brain-development risk options" range — phenylpiracetam's brain-dev profile is benign at PRN dosing.
  • Interaction risks:
    • Additive with other DAT inhibitors (modafinil, methylphenidate, amphetamine, cocaine, bromantane). Avoid same-day stacking — sympathetic + DA tone goes additive, with risk of overstimulation, hypertension, anxiety, sleep wreck.
    • MAOIs (theoretical): DA reuptake inhibitor + MAO inhibition could theoretically produce hypertensive episode. Selegiline at 1-2.5 mg (MAO-B selective) is probably fine; non-selective MAOIs are not relevant to the user's plan.
    • Caffeine: mild additive — usable at low caffeine doses (<100 mg) but stacking 200 mg phenylpiracetam + 200 mg caffeine + 200 mg L-theanine is overshoot for most users.
    • L-tyrosine: theoretically synergistic (more substrate for DA synthesis when reuptake is blocked) — anecdotal reports mixed.
    • Choline (alpha-GPC, CDP-choline): synergistic, prevents headache.

References

Wikipedia — Phenylpiracetam

en.wikipedia.org

comprehensive overview, mechanism, pharmacokinetics, regulatory status, key research history.

View Study

Wikipedia — MRZ-9547

en.wikipedia.org

pure R-phenylpiracetam, Merz development for Parkinson's fatigue, program status.

View Study

Zvejniece et al. (2017) — S-phenylpiracetam, a selective DAT inhibitor (PubMed 28743458)

pubmed.ncbi.nlm.nih.gov · 2017

molecular pharmacology, DAT IC50, body weight effects of S-enantiomer.

View Study

Savchenko et al. (2005) — The phenotropil treatment of the consequences of brain organic lesions (PubMed 16447562)

pubmed.ncbi.nlm.nih.gov · 2005

99-patient encephalopathy trial (commonly cited as "the 400-pt RCT" but actually 99 patients open-label).

View Study

Firstova et al. (2011) — Effects of scopolamine and phenotropil on rat brain neurotransmitter receptors

link.springer.com · 2011

receptor density data: D1 +16%, D2 +29%, D3 +62%, BZ +25%; NMDA + AChR density modulation.

View Study
Was this helpful?
Your feedback shapes what we research deeper.

How was your experience with this compound?

Anonymous · one vote per session · results below at 5+ votes.

Loading…

See something off?

Most of this wiki is AI-generated. Suggest a correction, dosing update, or new evidence — we review every submission.

Discussion — click to load
Loading…
Continue: Extended research →
Our verdict, decision matrix, deep dives, controversies, sources